The protocol was approved by the Animal Welfare Office of the Justus-Liebig-University Giessen
ermine whether CaMKII inhibition could prevent or reduce cardiac hypertrophy, we crossed mutant mice...
Reasons for this discrepancy are currently unclear
guanosine nucleotides by treatment with mycophenolic acid also resulted in increased susceptibility to AmB,...
It is also possible that free Ku70 could contribute to the Ku80-mutant phenotype
ild-type and mutant mice expressing AC3-I compared to mice expressing AC3-C. However, increased pCaMKII-T286/CaMKII...
The Wnt pathway has been activated by X-rays and dioxin
ients in the interruption group were younger than those in the persistence group. The...
However, the etiology of mitochondrial dysfunction is not known
ivate caspases that subsequently regulate nuclear export of the viral RNP complexes. Therefore, we...
Hnf4a deficiency increases both active signatures and repressive signatures at the same loci
iluted 1:200 for 20 min and with peroxidase for 10 min. Diaminobenzidine was used...
We were able to identify the toxin transcripts in all versions by RT-PCR
l Centre, Malaysia. This study was conducted in accordance to Declaration of Helsinki and...
The full length cDNA was amplified and confirmed after sequencing
this may enhance their susceptibility to CB1-mediated inhibition of intrinsic excitability. In addition, while...
For this purpose, we compared the neurite length of sAPPa-treated neurons and control ones
for eotaxin, CXCL1, and TNF-a was 3.0, 2.0, and 8.0 pg/ml, respectively. MMP-2 and...
The isolated live muscle fibers were incubated with TMRE to visualize mitochondria
oad is not decreased, resulting in the absence of negative staircase effect. Adrenergic Signaling...